Angioedema Congress Book by Georg Kojda, Murat Bas

By Georg Kojda, Murat Bas

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J Am Coll Cardiol 1998; 32: 1787 – 1796 Xu J, Carretero OA, Liu YH, Shesely EG, Yang F, Kapke A, Yang XP. Role of AT2 receptors in the cardioprotective effect of AT1 antagonists in mice. Hypertension 2002; 40: 244 – 250 Campbell DJ, Krum H, Esler MD. Losartan increases bradykinin levels in hypertensive humans. Circulation 2005; 111: 315 – 320 LeFebvre J, Shintani A, Gebretsadik T, Petro JR, Murphey LJ, Brown NJ. Bradykinin B2 receptor does not contribute to blood pressure lowering during AT1 receptor blockade.

AT1 receptor antagonists inhibit very selectively the AT1 receptor and completely prevent activities mediated via this receptor. Through prevention of the angiotensin IImediated inhibition of renin liberation, the formation of angiotensin II is increased which, via a stimulation of AT2-receptors, can lead to de novo synthesis of bradykinin and a stimulation of B2 receptor. , by inhibition of ACE is, in contrast, the subject of dispute. ACE inhibitors inhibit the formation of angiotensin II and suppress AT1 and AT2 receptor-mediated activities.

Rosenkranz1, W. Fan2, J. Zimmermann2 1 2 corresponding author: Department of Medicine, Division of Pharmacology, University of Stellenbosch, Cape Town, South Africa Jerini AG, Berlin, Germany Abstract The kallikrein-kinin system is an endogenous metabolic cascade responsible for the production of vasoactive kinins such as bradykinin. Pharmacologically active kinins are implicated in many physiological and pathological processes, such as vasodilation, vascular permeability, inflammation, pain perception and cardioprotection.

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